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A CLU/APOJ GWAS AD risk variant suppresses the astrocytic response to plaques and reduces axonal and neuritic damage in 5xFAD mice

Early occurrences in CSCC, such as ultraviolet-induced P53 mutations, cause significant genomic instability

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These events mitigate cellular reductive stress by suppressing key metabolic pathways, indicating a crucial role of L-2-HG

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