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Activation of the GLP-1R by GLP-1 also results in recruitment of -arrestin-1 (Arr1) and -arrestin-2 (Arr2), which mediate receptor internalization and may serve as scaffolds for G protein-independent signaling ( 18, 19 )

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Conclusion This study clarifies the key role of dopamine and D2 receptors in the mOFC in chronic migraine: in CM model mice, dopamine release in the mOFC is increased, which activates dopamine D2 receptor and inhibits excitatory DRD2+ neurons dominated by Camk2a, thereby improving pain hypersensitivity and anxiety-like behavior

[5] GURNEY, T., BROUNER, J., SPENDIFF, O

Moussa, S