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2 Mechanism of cisplatin ototoxicity Cisplatin enters the cochlea, mainly through the blood-strial barrier and the blood-perilymph barrier ( 2.1 Cisplatin-induced DNA damage and apoptosis Once cisplatin enters the cell it can be hydrolyzed to form toxic aqua-cisplatin complexes and form intra-and interstrand crosslinks with DNA (Figure 1) FIGURE 1 2.2 Cisplatin induces oxidative stress The cochleas high metabolic rate results in its production of abundant reactive oxygen species (ROS), so it requires an active antioxidant defense system [glutathione (GSH), oxidized glutathione (GSSG), and antioxidant enzymes (superoxide dismutase (SOD), catalase (CAT), glutathione peroxidase (GPx), and glutathione reductase (GR)] to achieve a dynamic oxidant-antioxidant balance to maintain healthy hearing

TB-500s appeal is the opposite: a clean, well-defined mechanism centred on a single, well-characterised protein family

The amount delivered per spray depends on the spray device, solution concentration, and consistency of administration

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Comparison of the effects of nicotinic acid and nicotinamide degradation on plasma betaine and choline levels