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First, its interplay with lipid metabolism: surplus pyruvate generated through glycolysis is converted to acetyl-CoA, which can enter lipid biosynthetic pathways and promote the synthesis of fatty acids, particularly PUFAs

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Bandsma et al

723 Mitochondrial dysfunction in senescent cells impair oxidative phosphorylation, increase ROS production, and disrupt fatty acid oxidation, leading to lipid accumulation and promoting steatosis and diseases such as NASH

doi: 10.1073/pnas.0610923104 263 SpencerP