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To confirm if the reduction in oxidative stress was biochemically significant, we quantified lipid peroxidation by immunostaining for 4-hydroxynonenal (4HNE) and observed a significant increase in hepatic 4HNE in mice on ethanol, which was suppressed by GLP-1R agonist treatment (Fig

10.1038/369756a0 11 BurcelinR

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Oxidants can stimulate many upstream kinases in signaling pathway cascades, and yet inhibit transcription factors AP-1 and NFB

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This system also contributes to the elimination of metabolic waste such as bilirubin