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4 The most influential primary work is a 2008 Gastroenterology study by Dalmasso and colleagues, which showed that KPV is transported into intestinal epithelial cells via the peptide transporter PepT1 and, once inside, reduces pro-inflammatory signaling

96,97 In turn, altered neural activity can further influence transcription factors or chromatin remodelling by transmitting action potential cascades that trigger signals and initiate specific transcriptional programmes

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Particularly, elevated levels of circulating pro-inflammatory mediators have been found in patients with treatment-refractory depression (TRD), including TNF-, IL-6, IL-1, CRP, and macrophage inflammatory protein-1 (88)