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While acute iron exposure produced minimal biochemical changes and did not sensitize cells to oxidative or ferroptotic challenges, chronic iron exposure induced ferritin upregulation, mitochondrial superoxide accumulation, suppression of GPX4 expression, elevated lipid peroxidation and loss of cellular glutathione (GSH)

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Frontiers in Endocrinology, 12, 585887

43.2.3.3 Glutathione Conjugation and Acetylation Glutathione S -transferases (GSTs) and N -acetyltransferases (NATs) together contribute approximately 25% of the phase II metabolic activity