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We hypothesize that adding a long-acting GLP-1-RA can increase basal GLP-1 levels and, in combination with native post-prandial GLP-1 bolus restored after MBS, may be considered as a basal-bolus treatment for individuals with MBS failure (Fig

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The persistent and recovered PTSD groups were included as dummy variables within the model

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At 36 weeks, a similar trend was observed but with minimal changes in HbA1c levels beyond 24 weeks in all groups [4]