Role of oncometabolites in modulating the epigenetic landscape of cancer cells Concerning the epigenetic alterations associated with oncometabolites, it has been shown that D-2HG accumulation, an event that is coupled with -KG depletion, inhibits histone lysine demethylases (KDMs), giving rise to histone hypermethylation, epigenetic programming, and acquisition of additional mutations [27]
4D and Supplementary Fig
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Stimulants include: S6.A: NON-SPECIFIED STIMULANTS Adrafinil Amfepramone Amfetamine Amfetaminil Amiphenazole Benfluorex Benzylpiperazine Bromantan Clobenzorex Cocaine Cropropamide Crotetamide Fencamine Fenetylline Fenfluramine Fenproporex Fladrafinil (2-[Bis(4-fluorophenyl) methylsulfinyl]-N-hydroxyacetamide) Flmodafinil (2-[Bis(4-fluorophenyl) methylsulfinyl]acetamide) Fonturacetam [4-phenylpiracetam (carphedon)] Furfenorex Hydrafinil (fluorenol) Lisdexamfetamine Mefenorex Mephentermine Mesocarb Metamfetamine(d-) p-methylamfetamine Modafinil Norfenfluramine Phendimetrazine Phentermine Prenylamine Prolintane A stimulant not expressly listed in this section is a Specified Substance
