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In vitro stimulation of human and murine macrophages and monocytes have demonstrated a proinflammatory effect of G6PD overexpression and a corresponding dampened response with G6PD-d 110

The H2S-dependent BP-reducing effect is thought to be primarily mediated through sulfhydration of ATP-sensitive potassium (KATP) channels, which in turn leads to voltage-sensitive channel opening and relaxation of vascular smooth muscle cells

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doi: 10.1128/AEM.02676-14