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Preclinical information from in vitro and in vivo studies of acute injuries and neurodegenerative disorders, as well as clinical practice concerns, are reviewed in conjunction with ferroptosis-targeted therapies

That extrapolation is scientifically reasonable but not yet validated by a clinical trial

Defective proteasome activity is detected in the early phase of AD along with synaptic dysfunction, and in late AD stages is associated with the accumulation and aggregation of ubiquitinated (Ub)-proteins which precedes tangle formation (Garca Gil et al., 2001

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The pathological mechanisms linking proteopathology and neuronal death have not yet been well characterised

Periodontal wound healing and regeneration