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Perhaps its most famous role, GLP-1 crosses the blood-brain barrier to act directly on appetite centres in the brain, enhancing feelings of fullness and reducing hunger signals

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Research conducted on obese mice has indicated that semaglutide treatment can reverse gut microbiota dysbiosis, particularly by increasing the abundance of beneficial bacteria, such as Akkermansia muciniphila

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While individual cases cannot establish causation, they reveal patterns that clinical trials may miss due to their structured reporting methods