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doi: 10.1056/NEJMoa1901118 20 PieberTRBodeBMertensAChoYMChristiansenEHertzCLet al
These effects were accompanied by increased NRF2-mediated transcription of brain-derived neurotrophic factor (BDNF) and decreased levels of methyl-CpG binding protein 2 (MECP2), a negative regulator of BDNF production (Tang et al., 2022)
The transition from oxidative phosphorylation to glycolysis directly precipitates an energy crisis within the heart, leading to contractile dysfunction, intracellular acidosis, enhanced oxidative stress, and cardiomyocyte death (52)
They can adopt various phenotypes, such as the inflammatory-promoting M1 subtype and the inflammation-suppressing M2 phenotype, and secrete different inflammatory mediators in response to inflammation [102,103,104]
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