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This analysis highlighted the unique binding potential of exendin 2029, suggesting its efficacy as a targeted TRPV1 blocker derived from exendin 939

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Furthermore, its vasodilatory effect may not solely depend on the increase in NO but could also involve other vasoactive substances, such as the reduction of angiotensin II or endothelin-1 activity, which promote vasodilation (162)

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The closure condition imposes the proliferation rate of each phase to be the opposite of each other