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Specifically, the wound area was significantly narrower, and the gap distance was significantly reduced after the 24 h of peptide treatment

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This agent is very promising for future clinical use since it perturbs the self-sustaining activation loop of JNK, instead of inhibiting JNK directly, and therefore would not be expected to inhibit the possible protective/beneficial effects of transient JNK activation in AILI [5]

Following 6 wk of ethanol administration, GCLM knockout mice were resistant to alcohol-induced steatosis and exhibited beneficial metabolic and stress responses to chronic ethanol consumption