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Mechanistic insights from non-AD neuroinflammatory systems provide complementary evidence: interferon- (IFN-) robustly induces PD-L1 expression in human astrocytes and microglia, establishing a checkpointredox interface that suppresses excessive oxidative injury (Smith et al., 2023

During seed germination, a delayed degradation of PSVs was also observed in the snx1 mutant and the snx1/snx2a/snx2b triple mutant (Fig

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