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In response, the brain elicits a neuroprotective, purinergic-receptor-dependent inflammatory response characterized by meningeal neutrophil swarming and microglial reconstitution of the damaged glial limitans
In this context, those effects are mediated via extracellular vesicles secreted by virally infected cells that contain viral effectors that modulate the metabolic activity of non-infected cells in the microenvironment
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Glucose promotes stress resistance in the fungal pathogen Candida albicans
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