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Although the exact mechanisms by which GLP-1 receptor agonists mitigate ischemic injury are not fully understood, the pleiotropic mechanisms seem to extend beyond glycemic regulation, with beneficial effects observed in nondiabetic animal models, wrote first author Michael K

Several large-scale RCTs and meta-analyses have reported no statistically significant increase in pancreatitis risk associated with GLP-1 RAs

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In vascular endothelium, GLP-1R stimulation activates both cAMP/PKA and PI3K/Akt signaling cascades, leading to phosphorylation of endothelial nitric oxide synthase (eNOS) and increased nitric oxide production [4]