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Hv1 inhibition rescues AD pathology by restoring microglial mitochondrial function and enhancing mitochondrial transfer
NRF2 also cooperates with components of the transcriptional machinery (i.e., MED16) (Sekine et al, 2016), chromatin remodelers (i.e., BRG1) (Zhang et al, 2006) and chromatin-modifying enzymes (i.e., CBP) (Katoh et al, 2001) to drive target gene expression
However, the unprotected oral glutathione actually reduced the active glutathione amount by 40 points, creating a 78-point difference between the two groups
While GLP-1 physiology informs the development of therapeutic agents, the native peptide itself cannot be practically administered for disease management
