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In aged skeletal muscle models, NNMT inhibitor treatment increased proliferating/active muscle stem cells by 60-75% following injury, myofiber cross-sectional area increased 1.8-fold at one week post-injury with high-dose treatment, and peak torque of injured muscle increased by 67% compared to control (p = 0.033), with no untoward systemic toxicity observed in plasma chemistry markers (Neelakantan et al., 2019)
Schmitt, A
& Lalk, M
Minimal adverse effects observed in preclinical models
These results replicate the intracellular A deposits previously reported that began in middle-age 3xTg-AD in vitro [1], and in vivo [38]
This phenomenon occurs because the Pi3K/mTOR pathway constitutes one of the key intracellular signaling pathways [115]
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