Antioxidant therapy as a potential approach to severe influenza-associated complications
This section discusses this common pathology by dividing it into four interconnected mechanistic axes: (i) a shared fibrosis-inflammation signal axis, (ii) the formation of a fibrotic microenvironment due to the disruption of immune homeostasis, (iii) oxidative stress-based microenvironmental lock-in, and (iv) upstream regulation failure due to barrier dysfunction
Ann Hematol 98:13331339 Coskun E, Keskin A, Suzer T et al (1998) Spinal cord compression secondary to extramedullary hematopoiesis in thalassemia intermedia
Mean blood pressure (MBP, mmHg) was monitored continuously by a Haemosys computerized dataacquisition system through the femoral catheter using a pressure transducer connected to an electromanometer (Experimetria Ltd., Budapest, Hungary)
Overexpression of glutathione peroxidase increases the resistance of neuronal cells to A beta-mediated neurotoxicity
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