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The probable pathway likely involves PCs augmenting GLUT4 translocation and glucose uptake in skeletal muscle through an AMPK-dependent mechanism, subsequently enhancing GLP-1activated insulin signaling and lowering postprandial hyperglycemia

Were going to say yes, but on this one

Evidence Summary Electrolyte disturbances are typically secondary to gastrointestinal side effects rather than primary drug action

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These could include lymph node mapping, imaging scans and a blood test to check for an enzyme called lactate dehydrogenase or LDH

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