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While pathogens directly disrupt mitochondrial homeostasis through toxins, viral proteins, or metabolic hijacking, the ensuing immune activation further amplifies mitochondrial dysfunction

doi: 10.3390/ijms19113306 16 KullmannSHeniMHallschmidMFritscheAPreisslHHringHU

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however, unlike classical ligand-regulated receptors, NHR-49 is regulated through ligand-independent mechanisms involving cofactor-mediated transcriptional attenuation and protein turnover

Vasodilatory effect Studies showed that GIPR and GLP1R are present in the smooth muscle cells and endothelial arterioles and may have a cardioprotective effect due to endothelium-dependent vasodilation [105]