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It is investigated in experimental models for its interaction with growth factor signaling, angiogenesis-related mechanisms, and cytoprotective processes

It is primarily produced in the central nervous system, and was first isolated from the brains of rabbits that were subjected to sleep by electric stimulation of the intralaminal thalamic area

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Mutations in homocysteine metabolism genes increase keratin N-homocysteinylation and damage in mice

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As indicated, despite the tumor-promoting effects of many growth factors and peptides (Sikiric et al., 2018), BPC 157 has been shown to inhibit and counteract uncontrolled cell proliferation (Ki-67 overproduction counteracted) and increased expression of VEGF and subsequent signaling pathways (Radeljak et al., 2004