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doi: 10.1016/S0098-2997(02)00016-X

[DOI] [PMC free article] [PubMed] [Google Scholar] 26.Exner R., Wessner B., Manhart N., Roth E

These differences likely reflect complex interactions among region-specific neuronal subtypes, glial responses, genetic predispositions, and disease-specific triggers, each contributing to unique patterns of redox imbalance

This is why a thorough medical history review is essential

These mitochondrial protein adducts are thought to cause mitochondrial dysfunction and to promote oxidant stress

Introduction Liver dysfunction leads to metabolic disorders and ultimately endangers personal health ( NAFLD is a chronic progressive lesion involving in inflammation, oxidative stress, insulin resistance, and imbalances in lipid metabolism ( Several studies have demonstrated that ferroptosis plays a crucial role in the occurrence of NAFLD ( Ferroptosis and non-alcoholic fatty liver disease/non-alcoholic fatty liver disease-hepatocellular carcinoma Ferroptosis Ferroptosis is a non-apoptosis with a completely different morphology