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Previous studies demonstrated that BP NSs effectively scavenge excess ROS by oxidizing its phosphorus atoms to ameliorate atherosclerotic plaques 21,22 , which suggested that BP NSs could be a ROS-stabilizer for CeO x -based nanozyme via self-oxidation to avoid the demolition of the Ce (III)/Ce (IV) redox cycle and improve its ROS-resistant durability in the microenvironment

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Cancer has evolved several regulatory mechanisms of ferroptosis, which neutralize ROS and prevent lipid peroxidation, including the xCT-GSH-GPX4 axis [15,42,44] , ferroptosis-suppressor-protein 1 (FSP1)-coenzyme Q10 (CoQ10)-NADPH axis [87] , and Hippo pathway [88]

Once inside the mitochondria, these fatty acids are oxidized to produce energy in the form of ATP (adenosine triphosphate), a molecule that fuels many cellular functions