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MYC promotes hepatocarcinogenesis in chronic liver disease and overexpression is associated with poor response

PLoS One 10, e0121257

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P53 (Ad-p53)

The activity of several involved enzymes also increase, including those that allow an influx of sulphur (as inorganic sulphate, that is converted into homocysteine as the entry step into biological organic species), which is the rate-limiting element for the biosynthesis of biological thiols [25,26]

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