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Differential aetiologies of hypertension arise from complex interplays between genetic and environmental variables that modify biological pathways and lead to the disease (124)

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found that early menopause cases had higher tumor necrosis factor receptor 2 (TNFR2) levels than controls (Bertone-Johnson et al., 2019)

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value obtained from CBC) resulting in an intracellular concentration of mole L 1 for GSH and GSSG

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