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Given that obesity is a hallmark of BBS and a significant driver of its associated comorbidities, a deepening understanding of BBS5s role in metabolism could yield critical insights into disease pathogenesis

Contrasting roles of NADPH oxidase isoforms in pressure-overload versus angiotensin ii-induced cardiac hypertrophy

Understanding this broadens the perspective

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Finally, we administered ASO 56 to the PSC mouse model, Mdr2KO mice, and measured liver inflammation and fibrosis

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