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The activation of PLC results in the release of inositol-1,4,5-trisphosphate (IP 3 ) from membrane associated phosphatidylinositol-4,5-bisphosphate (PIP 2 ), which then binds to specific IP 3 receptors in the endoplasmic reticulum/sarcoplasmic reticulum (ER/SR) resulting in the release of intracellular stores of Ca 2+

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Control group (EPPT without biomaterials) resulted in an amount of CAL gain (5.83 1.12 mm) and PD reduction (6.2 1.33 mm) which demonstrated a significant clinical improvement

TNF- caused mitochondrial mPTP opening, apoptosis, and downregulation of phosphatase and tensin homolog signal, resulting in excessive Bnip3-dependent mitophagy and mitochondrial dysfunction [79, 80]

doi: 10.1530/eje.0.1470077 107 WangTLuJXuYLiMSunJZhangJet al

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