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First, using germ-free animals, directional microbiota transplantation, and gene editing techniques to clarify the causal mechanisms by which protective genera such as Bifidobacterium regulate the hepcidin-FPN1 axis and key molecules of chondrocyte ferroptosis (GPX4, SLC7A11), thereby consolidating the logical chain of microbiota-iron metabolism-cell death. Second, leveraging longitudinal cohorts and multi-omics technologies to reveal the time-dependent evolution of the gut dysbiosis-iron overload-ferroptosis-intestinal dysfunction cycle and identify key windows for early intervention

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This study found a 35% greater weight loss difference in women combining hormone therapy with tirzepatide compared to tirzepatide alone

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These effects suggest possible roles in maintaining strength and vitality

Hyperhomocyst(e)inemia is a risk factor for arterial endothelial dysfunction in humans