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These differences probably account for remyelination occurring later in the cortex than in the CC: after acute cuprizone-induced demyelination, OPC repopulation and differentiation is much faster in the CC than in the cortex, resulting in later, incomplete remyelination in the cortex (Gudi et al., 2019)

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Methionine-loading experiments in control and folate-deficient animals indicated that homocysteine plasma concentration correlates with platelet aggregation and the biosynthesis of the powerful proaggregant thromboxane A 2 (Durand et al, 1997a)