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Importantly, this implies that FSP1 may be essential not only in cancer resistance to ferroptosis but also in physiological contexts such as adipose tissue metabolism, where regulation of neutral lipid oxidation is critical for maintaining tissue homeostasis
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, In our study, sulfasalazine administration inhibited the formation of TLS and attenuated the expression of TLS-related cytokines, with sustained tubular injury
doi: 10.1172/JCI89934 29
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