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Glucagon-like peptide-1 receptor agonists activate rodent thyroid C-cells causing C-cell hyperplasia and tumors
10.1111/nmo.12468 55
Nestler JE, Jakubowicz DJ, Reamer P, et al
Retrieved 2010-08-02
ika O, Haluzk M, Jude EB
Directly supplementing with oral reduced glutathione (L-GSH), however, presents a fundamental pharmacokinetic challenge: GSH has a plasma half-life of approximately 1.6 minutes, undergoes rapid hydrolysis in the gut by intestinal -glutamyl transpeptidase (-GT), and cannot cross cell membranes intact requiring full breakdown into constituent amino acids and intracellular resynthesis
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