The mechanism involves upregulation of growth hormone receptors in tendon fibroblasts and increased collagen synthesis at the repair site
The majority of the studies (see above in red) are preclinical studies in rodents, the vast majority of which have been done by a single research group in Croatia, which has been rinsing and repeating the same approach for decades
A short history: wound repair first, then skin and scalp care GHK-Cu got attention in early wound research because it showed signals tied to tissue repair, collagen organization, and reduced inflammation in skin models after injury
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