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The action of this probiotic was attributed to its ability to increase the levels of SCFAs (butyrate and propionate) via nicotinamide metabolism in the serum and kidney, with reduced renal inflammation and decreased damage to the renal tubular epithelial cells as the resultant effect (Zhu et al., 2021)

The majority of plasmatic GSH originates from the liver, and for this reason an impairment in hepatic GSH synthesis has a systemic impact on redox balance and oxidative stress (1, 6, 7, 11)

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