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The 48-h cARLA treatment resulted in mature brain endothelial cell morphology with continuous and strong claudin-5 junctional protein immunostaining at the cell borders (Fig

By disrupting the functions of these intracellular molecules, it may be possible to sensitize CSCs to conventional treatments, reduce tumor recurrence, and improve patient outcomes

All these data also suggest the important role of mtDNA mutations causing mitochondrial functional defect for tumorigenic process and emphasize the tumorsuppressive function of normal mitochondria

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Thus, the effect of CK1, PKC, and p38 MAPK varies from pro-survival in some erythrocyte precursors to pro-eryptotic in mature cells, which might be a consequence of missing downstream effectors in circulating erythrocytes