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Beyond the traditional crosstalk between the most recognized insulin-responsive organs (liver, adipose tissue, and skeletal muscle), kidney insulin action and resistance have recently been suggested as critical components of metabolic and dysmetabolic states (Musso et al., 2014

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Oxidative stress has the potential to intensify neuronal hyperactivity and excitotoxicity, particularly through the glutamatergic dysfunction and overactivation of glutamate receptors, consequently heightening susceptibility to epilepsy (Borowicz-Reutt and Czuczwar, 2020

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