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However, it is important to note that all molecules reacted rapidly at elevated pH, suggesting potential nonspecific interactions between the deprotonated Cys13 and the warhead of the nucleotides, irrespective of their binding to the nucleotide-binding pocket

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U S A 110, 22282233

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The resulting catastrophic energy deficit is a primary physiological driver of post-exertional malaise (PEM) or 'crashes,' where even minor, everyday activities trigger a disproportionate and prolonged exacerbation of profound fatigue, muscle weakness, and cognitive impairment