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Ferroptosis and Cell Death Mechanisms in Parkinson's Disease
Discussed earlier, NRF2-dependent upregulation of POMP is needed for hESC self-renewal and reprogramming and its expression decreases as hESCs become more differentiated ( NRF2 Controlled Redox Enzymes in Mediating Stem Cell Maintenance As NRF2 was originally identified as a master regulator of cellular antioxidant defense, it is not surprising that many of its identified target genes are involved in preventing and mitigating the harmful effects associated with increased oxidative stress
GSH, for example, can reduce H 2 O 2 , resulting in GSSG and water [99]
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