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Discussion In this study, we employed a two-sample MR analysis to explore the bidirectional causal relationships between LC-related subtypes, their metabolite levels, and SZ
S7) to clarify the relationship between reduced glycogen, GSH depletion, and increased susceptibility to APAP in hepsin / mice
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All mice were fed a nutritionally adequate liquid control diet for a week, and then divided into six groups (control, model, PGPIPN I, PGPIPN II, PGPIPN III and GSH) with 10 mice in each group
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Our roGFP2 measurements confirm (i) that dynamic roGFP2 sensing (or the physiological glutathionylation/deglutathionylation of regular surface-exposed cysteinyl residues) requires a glutaredoxin activity, and (ii) that glutathionylated proteins or metabolites should be very transient redox species when GSH and glutaredoxins are present (unless they are stabilized by a kinetic uncoupling mechanism) 5,32,34,35,61,62,63,64
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