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This decline suggests that pre-treatment with both antioxidant agents effectively suppressed carboplatin-induced apoptosis and inflammation

Thus, glutathione-mediated preservation of mitochondrial function, in our opinion, became possible due to the reduction of ROS and restoration in mitochondria of the synthesis of gas mediatorsNO and H 2 S which are important natural regulators of mPTP

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Front Endocrinol (Lausanne) 14:1234918

Disrupting metformin adaptation of liver cancer cells by targeting the TOMM34/ATP5B axis

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