POTENTIAL MECHANISMS UNDERLYING INFECTION-ASSOCIATED DYSAUTONOMIA & POTS The autonomic nerves or autonomic ganglia may be affected by: Chronic infection Autoimmune response triggered by an infection Damage to autonomic nerves by an infection Neuro-inflammation triggered by debris left-over after an acute infection is treated CHRONIC ILLNESSES ASSOCIATED WITH POTS Post Treatment Lyme Disease Long COVID Myalgic Encephalomyelitis/Chronic Fatigue Syndrome or ME/CFS Sjogrens Syndrome Systemic Lupus Erythematosus Hashimotos Thyroiditis Celiac Disease Diabetes MS Other Infection Associated Illnesses RESEARCH NEWS Review Article by Brit Adler, MD, in Frontiers in Neurology: Dysautonomia following Lyme disease: a key component of post-treatment Lyme disease syndrome
Rawat, Shalini et al (2022) RUFY1 binds Arl8b and mediates endosome-to-TGN CI-M6PR retrieval for cargo sorting to lysosomes
Recovery benefits like less soreness, better sleep, and steadier energy for workouts often show up by the next day or next training session
The activity of both WNK1 and WNK4 are increased in the presence of high dietary potassium which results in the inhibition of NCC and the activation of ROMK
In addition, these patients also showed signs of immune and endothelial dysfunction, elevated oxidative stress, inflammation, and impaired mitochondrial function
Various enzymes in the PPP have been shown to be potential targets in cancer therapy
