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Analgesic amounts stimulated respiration via central mu1 receptors, cataleptic amounts depressed breathing and blunted the CO2 response, and the peptide lowered blood pressure documenting the dose-dependent opioid effects including respiratory depression

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further received research funding from the German Research Foundation (TRR152 and TRR296)

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BPC-157 stimulates VEGF expression and VEGFR2 signaling, which drives the growth of new vascular networks directly into the injury site, dramatically increasing the delivery of oxygen, nutrients, and healing cells