doi:10.1152/ajpheart.00416.2017 Colman R, Tchkonia T, Pirtskhalava T, et al
Normalization by post-treatment cell counts was intentionally avoided to prevent the mathematical masking of the total oxidative stress that drives cell death
116 Thus, it appears that the decrease in sGC expression elicited by high blood pressure is not exclusively linked to oxidative stress and/or to increased levels of vasoconstrictor peptides
A decrease in the antioxidant capacity of the brain may have functional consequences through cell damage, chronic inflammation, protein, and DNA damage
03/02/15 - Removed section on parallel vs
Many insights come from studies exploring how metalorganic complexes interact with cellshow they enter them, what redox states they adopt, and how they influence signaling pathways