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It can turn on survival promoting signaling cascades, amplify inflammation, act mitogenetically against therapy-induced oxidant pressure, or stimulate ferroptosis, a non-apoptotic necrotic-like morphing of deceased cells marked by iron-dependent lipid peroxidation [16, 21,22,23]

The ROS disrupt essential cellular processes by interacting with DNA, RNA, and protein synthesis, which may result in inflammation, cell necrosis, or apoptosis 14

The impact of glucagon-like peptide-1 receptor agonists on kidney outcomes: a meta-analysis of randomized placebo-controlled trials

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Both MS pathology and DMTs appear to affect circulating erythrocytes

The combination is described in pharmacology literature as synergistic