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While the phenotypic insulin resistance in NAFLD would be expected to counteract insulin-mediated SREBP1c activation, a state of selective insulin resistance ensures that insulin retains its ability to promote DNL through SREBP1c while being unable to suppress gluconeogenesis [41]

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It can serve as a reservoir of reduced sulfur, interfacing with sulfur metabolism and biogenesis of sulfur-containing cofactors

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In both cells lines, the wt strain, as well as and the gpoA, tpxD and ahpD mutants, showed an increased intracellular survival caused by the low ROS levels after the NAC treatment (Fig

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