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Clin Pharmacokinet 11:250256 Forrest JAH, Adriaenssens P, Finlayson NDC, Prescott LF (1979) Paracetamol metabolism in chronic liver disease

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Finally, we review mechanismbased strategiesincluding KRAS G12C inhibitors, effluxpump antagonists, apoptosis reactivators, and epigenetic/autophagy modulatorsand propose an integrated, multimodal regimen leveraging immunotherapy where appropriate, informed by real-time drug sensitivity data (e.g., from liquid biopsy), dynamic biomarkers and AIdriven optimization to overcome resistance and improve patient outcomes

It exists as an active dimer 45 and targets phosphorylation of downstream targets such as CHK2, H2AX and p53 44,47,48,49