demonstrated that GIPR agonism could reduce the risk of atherosclerosis in sensitized animal models, and human genetics revealed a correlation between hypoglycemic alleles of GIPR and reduced BMI, triglyceride levels, and heart failure incidence [51]
Compared to the saline, MCMs, and SP groups, the SP@M treatment could satisfactorily increase the maximum contact and print area, even higher than that of the MTX treatment (Supplementary Fig
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