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The HGF/c-Met pathway activated by Dihexa promotes neuronal survival through activation of PI3K/Akt signaling cascades that inhibit apoptosis pathways and enhance cellular stress resistance

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We postulate that ER signaling confers ferroptosis resistance by transcriptionally upregulating antioxidant defenses such as glutathione peroxidase 4 (GPX4), a mechanism recently validated in other epithelial models [35]

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